USING NEUROFEEDBACK TO CORRECT THE INCORRIGIBLE
George von Hilsheimer and Douglas A. Quirk*
ABSTRACT
Quirk learned to apply Mary Cover
Jones' method of desensitization to psychotics despite Wolpe's
self-reported inability to make Reciprocal Inhibition Therapy
(RIT) work with psychotics. Von Hilsheimer suggested that he monitor
stress by GSR and Quirk developed a library of lantern slides
to substitute for verbal statements in the hierarchies of stressful
stimuli. He automated this method and named it SCARS (Stimulus
Conditioned Autonomic Response Suppression)
Quirk then applied the same methods
and Sterman's EEG biofeedback training (increasing SMR at C-3/C-4)
in a pilot study of 40 matched pairs of jailed felons; and in
a larger pilot study of 110 matched pairs. From 1970 through 1995
Quirk trained 2776 felons by this combined method (temperature,
GSR and EEG)
15% were rearrested in the 3 years
following release. This compares well to the range of rearrest
in studies summarized by Alter et al (1996) - 42 % to 78%.
INTRODUCTION ICTAL AND SUBICTAL DYSFUNCTION
Two of Quirk's colleagues, North
and Breen, liked a method of scoring the Bender-Gestalt published
in a book by Hutt. However, the Bender figures weren't really
adaptable to the Hutt method. So North and Breen created new figures
and called the test the Diagnostic Differential Test (DDT). Quirk
took a course in administering the DDT and became expert at its
interpretation.
One of the neurologists on staff
who used Doug as a consultant gave him a list of 10 patients whose
serial EEGs eventually demonstrated epilepsy but who did not have
seizures. He was asked what test to use to figure out how to recognize
them more reliably than with the EEG. Doug found a file into which
he had put the DDT's those individuals' had taken. Quirk had considers
those particular DDT's as uninterpretable.
About the same time Quirk had been
wandering around the halls at McGill on a trip to Montreal and
had a casual conversation with a psychologist who told him of
some work he had been doing with pigeons demonstrating that when
an ablation was made in the region of the brain's septum the pigeons
that had been trained to respond to angular signs were post operation
unable to differentiate the angles from curves.
The performance of his 10 puzzling
patients on the DDT indicated that they couldn't handle angles
as well as they could handle curves. So Quirk decided that he
had found an indicator of deep diencephalic epilepsy. The first
70 patients he found with this sign on the DDT were all diagnosed
by a neurologist as epileptic. All but one had typical seizure
spindles on the EEG.
Quirk applied the DDT to a large
number of arsonists, assaultists and rapists. Forty per cent of
the serious arsonists, 30% of assaultists, and 25% of rapists
demonstrated the sub-ictal sign on the DDT. This last group, the
rapists with anomalous DDT performance, also demonstrated visible
anomalies of the temporal lobe in CAT scans.Quirk's insight that
a great deal of maladaptive behavior might be epileptoid was reinforced
by Jonas in ICTAL AND SUBICTAL NEUROSES, and by Alvarez in NERVES
IN COLLISION.
Quirk moved from Queen Street Hospital
to the Clark Psychiatric Institute in Toronto around 1965 where
he continued work using the SCARS GSR biofeedback we had earlier
developed refining the process by which SCARS works.
Quirk was a pioneer in the
use of volunteer workers in psychotherapy and most of the procedures
he used were administered through automatic equipment monitored
by volunteers. In his setting the doctoral level staff were free
to diagnose and order treatments which worked.
BARRY STERMAN
In 1970 Quirk went to a meeting
in Boston and heard Barry Sterman talk about his work with cats
and human epileptics. Doug had lunch with Barry and on Sterman's
recommendation bought an Autogen 120a EEG feedback device
From the Clark Quirk moved to the
Ontario Correctional Institute (OCI) where he has spent more than
20 years improving an excellent facility. OCI had the lowest recidivism
rate in the Western World.
In several years the OCI was recognizedby
professional correctionists as the "best" correctional
facility in North America
CRIMINALS IN JAIL
Quirk completed two preliminary
studies of recidivism among OCI felons who had been discharged.
He compared 55 pairs of high risk felons half of whom were treated
by temperature biofeedback, by SCARS and by Sterman's EEG method
and half of whom received only counseling. These violent felons
demonstrated the subictal pattern on the DDT (viz., they didn't
handle visual angles as well as visual curves) and on the EEG.
The treatment of the 55 felons included volunteer administered
temperature biofeedback training; SCARS GSR training; and EEG
training of the sensorimotor rhythm at C-3 and C-4 as described
by Sterman. These felons received 33 half-hour sessions of training.
Subsequently he trained another group of 55 pairs of felons
Two years after his initial study
of 55 pairs of felons 22 or 40% of the treated felons had been
arrested again. Two years after the second study of 55 pairs of
felons 11 or 20% of the treated felons had been rearrested. This
rate of recidivism compared to 85% and 65% rearrested among the
matched felons who were not treated by biofeedback. In other words,
60% and 80% of the treated felons were still free, while only
15% and 35% of the untreated felons had managed to remain outside
of prison.
Quirk treated 150 more felons using
the same method of SCARS and SMR neurotherapy, matched with felons
receiving only counseling. We have been unable to verify the data
on recidivism in this last study.An intriguing finding is that
recidivism is a function of the number of training sessions. Half
as many felons who were treated for 16 sessions were successful
at remaining out of jail as were those who received 32 sessions.
There is some effect of training even in a few treatment sessions.
In a careful study of 260 young
violent prisoners Quirk was able to demonstrate that it is possible
significantly to reduce recidivism from using relatively inexpensive,
volunteer applied techniques. In an earlier study of 150 women
whose average time on a closed ward had been 9 years Quirk demonstrated
that 128 could safely be discharged after using inexpensive, volunteer
applied biofeedback technique.
Quirk's elegant combination of
several hypotheses implies that our own success using milieu therapy
to remediate a different population of young felons may be enhanced
by structuring our own assessments more elegantly in terms of
a similar hypothesis as to the nature of the disorder which leads
adolescents into treatment centers. These EEG responsive disorders
appear to have a common characteristic in that they seem mediated
through the physical structures in the diencephalon within which
the limbic system functions. These disorders may be a special
form of partial seizure with complex symptomatology.
BIOFEEDBACK IN PRODROMELESS
PSYCHOSIS:CAN YOU FIX PATIENTS WHO GIVE NO WARNING?
INTRODUCTION
We are here concerned with
the applicability of biofeedback treatment to some major disorders
in which there appear to be no observable and no conscious prodromes
to cue re-institution of learned corrective habits.
In
some conditions, pain (e.g., migraine) or an aura (e.g., some
cortical epilepsies) signals the need to activate self-regulatory
behaviour which has been trained into a person by biofeedback.
In contrast to migraine and grand mal epilepsy certain psychomotor
seizures give nowarning at all, indeed the patient may not even
know that he has hada seizure when it is over. In these conditions
the afflicted person is unlikely to be aware of any cues that
signal the imminent onset of a deep brain subictal foci causing
bizarre behavior. These seizures used to be known as psychomotor,
or templar lobe epilepsies.
PARTIAL COMPLEX SEIZURES
Today they are more likely
to be called partial complex seizures (a shorthand which arised
from Partial Seizures with Complex Symptomatology, [PSCS]). These
subictal foci typically do not result in the motor seizures of
cortical epilepsies; but, they are sometimes associated with dangerous
criminal automatisms, or with the emergence of symptoms of some
schizophrenias. In undertaking biofeedback treatment of these
latter conditions, it may be necessary to adopt a different training
procedure from the self-regulatory 'informational' feedback most
commonly used as biofeedback.
In
one study the present writers tested for deep-brain partial and
complex seizures in the various types of offenses encountered
in one year's admissions to a correctional treatment facility.
We found that 40% of the arsonists, 30% of the assaultists and
25% of the rapists were subject to deep-brain paroxysms -- perhaps
mediated by accessory activation of reinforcement, rage and/or
sex centres of the drive centre.
By way of contrast, fewer than
2% of the remaining 'garden varieties' of less dangerous offenders
exhibited this deep-brain epileptic syndrome. In several other
studies we found that there was a consistent and strong relationship
between strong emotional reactivity or weak emotional control
and dangerous criminal actions.
BEHAVIOURAL CHARACTERISTICS
There
are several reasons for selecting dangerous criminal offenders
and schizophrenics as the subjects for the present study. The
first obvious reason is the importance of these two types of conditions
in society and in clinical psychological practice.
The second obvious reason is that
both of these conditions pose particular problems for therapeutic
intervention, and thus warrant study in the context of any emerging
technology. A third reason is that criminals and schizophrenics
have been found in many studies to demonstrate anomalous psychophysiological
measures. These physical measures of psychological processes might
be susceptible to modification by means of biofeedback treatment.
THE RELEVANCE OF CUES
Most
conventional biofeedback training involves relatively continuous
feedback tracking changes in the physiological responses being
monitored. One point of view concerning the efficacy of such training
is that the treated conditions are associated with detectable
stimuli, and that these stimuli serve as cues to arouse the subject
to reinstate the learned self-regulatory behaviour. Cuing stimuli
might include prodromes such as pain in migraines, aura in epilepsies,
the characteristics of settings such as a classroom or a verbal
text in attention deficit or dyslexia, or even just the passage
of time in meditation training, sleep regulation or chronic pain
control.
It
seems likely that most conditions possess some readily perceived
cues that might be used to activate learned corrective behaviour.
PARTIAL COMPLEX SEIZURES
Some conditions are such
that it would be extremely difficult for the subject to achieve
such a conscious awareness of the cue as to be able to trigger
a trained therapeutic response. This difficulty is probably most
true in the cases of complex partial seizures and of psychotic
disorders.
It
is generally agreed that some deep-brain complex partial seizures
are facilitated by alcohol ingestion. In these cases the act of
drinking an alcoholic beverage might serve as a cue if the corrective
biofeedback training were to be undertaken in the context of drinking.
Failing this, the use of alcohol would come to serve as a cue
only if the subject undertakes the necessary anticipatory training
on her own. Otherwise, the patient has to rely on some other kind
of cue.
The
most common prodrome in deep-brain complex partial seizures seems
to be heightened intensity of emotional arousal -- perhaps sometimes
due to associated activation of the Olds and Milner 'drive centre'.
The difficulty encountered by most people in using strong emotional
arousal as a cue for behaviour is that emotional arousal tends
to distract attention from conscious cortical self-regulatory
habits. High emotional intensity tends to disengage the subject
from such moderating habits. Distracted attention and disengagement
is intensified by alcohol, by recreational and medical drugs,
all of which are often involved in scenes of intense emotionality.
Stated differently, emotional arousal
is mostly pretty confusing and, if intense emotion activates any
habits, the habits engaged tend to be ones which were learned
early in life and are relatively rigid habits in contrast to the
more recently learned adaptive and regulatory habits. The patient
learned to be disruptive as a child, what the therapist is ableto
train in the adult is often weak and ineffective.
ISN'T IT A DISEASE? You Can Treat It And Cure It!
In
1967 von Hilsheimer published IS THERE A SCIENCE OF BEHAVIOR?
reviewing particularly the success of replication therapies among
the behavior therapies. Quirk had already been demonstrating the
utility of our automated GSR biofeedback method with psychotic
patients. By 1971 Quirk had dramatically demonstrated the effectof
temperature and GSR training on schizophrenics who had beenin
hospital for 2-45 years (average 9).
Both
of us remarked that a consistent problem occured when working
with dangerous criminals and with young patients whose early delinquency
later proved to be a prodrome for psychosis. This problem was
the absolute absence of a prodrome for destructive explosive episodes
of behavior.
Clinicians
of great intuitive skill were helpless when it came to a type
of criminality for which no one had a solution. The common rate
of recidivism in correctional institutions in the United States
was generally believed to be 98% within two years of discharge.
Prisons are so bad that it could be said that we were failing
to criminalize only 2% of the juvenile population enrolled in
our higher institutions of learning (correctional institutes).
It happened that this general belief (even general in the correctional
professions) was not true, but that is another essay (Alter op
cit).
ANOTHER METHOD FOR REHABILITATING
CRIMINALS: EEG
Joe Kamiya's extention of
Shagass' earlier demonstration that electroencephalographic (EEG)
activity could be altered and deliberately shaped by feedback
of that activity, launched EEG biofeedback as a clinical reality.
Kamiya's demonstration also suggested that the electrical activity
of the brain is just a kind of behaviour that can be trained like
any other. Let's phrase that in another way. We can condition
the electrical activity of the brain. If this is true then any
recognizable feature of the EEG whether it is pathological or
healthy, desired or undesired can be changed. If behavior is associated
with EEG activity then behavior can be changed by changing the
EEG.
The
recognition that brain activity is direct maleable was the seed
from which grew Forester's habituation training for triggerable
epileptics and Sterman's conditioning of sensorimotor rhythm (SMR)
to inhibit epileptic seizures. EEG conditionability also made
it possible to normalize learning performances of people exhibiting
attention deficiencies and hyperactivity. But how widely might
this idea be applied? (1) Some epilepsies, whose focus lies in
the deep recesses of the old brain, are variously referred to
as partial seizures with complex symptomatology (PSCS) or "complex
partial seizures". These are non-convulsive seizures, or
paroxysmal events, sub-ictal states, or seizure equivalents.
These deep-brain seizures
have features that make them difficult to recognize.
They
are hard to recognize, first, because sub-ictal states are seldom
associated with convulsions; partial seizures do not cause dramatic
losses of consciousness; the complex symptoms do not seem to have
prodromes or even periodic unusual events that might suggest the
need for a specific investigation (see Spiers, Schomer, Blume
and Mesulam, Chapter 8, Temporolimbic epilepsy and behavior, in
Principles of Behavioral Neurology, Mesulam, F.A. Davis Co., Philadelphia,
1985; Jonas, Ictal and SubIctal Neurosis and also see Alvarez,
Nerves in Collision).
Second,
these sub-ictal neuroses are not readily diagnosed by means of
the EEG, partly because they are not easily triggered, as the
cortical epilepsies can be triggered, by means such as photic
stimulation or hyper-ventilation.
Third,
the sub-ictal states or epileptic equivalents often seem to be
accountable as behavioural derivatives of the developmental and
social history of the criminal. Which is to say the victims often
seem just to be bad boys with a long consistent history of bad
behavior arising in a bad neighborhood with other bad boys.
Fourth,
the behaviours associated with these sub-ictal states tend to
distract the observer because they are intense, gruesome, unappetising
and usually criminal activities. The underlying disorder is simply
ignored. If the epileptic discharge in these seizures involves
the Olds and Milner 'drive centre', the person may exhibit uncontrolled
paroxysms of rage, sexual drive, hunger, satiety, alcohol use
or other excitant automatisms such as fire-setting. The individual
may seem perfectly normal in just a few seconds after the explosion
of aberant behavior.
It
is not too surprising that when you examine people in prisons
that you find that the correctional population contains relatively
large numbers of people exhibiting this paroxysmal syndrome; but
it is the criminality, rather than the pathology of the behaviour
which is most likely to attract the observer's attention.
If
Barry Sterman's procedure for SMR training of the EEG is effective
in treating some epilepsies, we thought it would be interesting
to discover whether or not it could be used in these deep-brain
partial and complex seizure states to modify their future criminal
conduct. If their criminality could be reduced then the value
of Sterman's method would be substantially greater than mere neurology
suggested. In SMR treatment, the training feedback tends to be
discontinuous and contingent on SMR occurrences -- that is, it
is operant training, rather than self-regulatory training. Consequently,
if SMR training reduces criminality, we reasoned that the SMR
learning could be said to have become a stable new habit requiring
no maintenance exercises. We could detect no recognizable prodrome
in these cases to serve as a cuing stimulus to re-activate any
learned self-regulatory strategy. So this self-sustaining response
is exactly what we needed.
Of
course the issue did not seem to us to be quite as simple as that.
Clinical experience with offenders exhibiting complex partial
seizures suggested that, whether or not it served as a recognizable
prodrome, emotional arousal did appear to function as a triggering
eventfor seizure equivalents in these people. There is a subclass
of criminal which is floridly emotional and that emotionality
is almost always associated with grotesque and extreme acting
out.
If
our observation was accurate and if we were to achieve a stable
corrective habit by means of SMR training, it seemed necessary
also to ameliorate the intensity of emotional arousal as part
of the treatment. Accordingly, we applied the same SCARS method
we had used with chronically hospitalized schizophrenic women
to our criminal males. That is we included discontinuous and contingent
training of the GSR at the same time we were using the EEG to
train SMR. Von Hilsheimer'sexamination of the records after Quirk's
death indicates that every singlefelon who received EEG training
had first been trained in the SCARS method of GSR training. Aside
from a few pilot cases where rather weak effects were achieved,
we did not perform a formal investigation of the effects of SMR
training alone on criminality.
Later,
von Hilsheimer (1977) was to discover that his stringently diagnosed
population of criminal psychopaths eventually emerged as chronic
psychotics and proved to be the most resistant of all populations
(including brain injured and chronically mentally ill patients)
to the markedly good effects of the milieu and behavior therapy
program he demonstrated (ibid).
We
have already discussed the development of the method of training
a group of chronic schizophrenic patients with a GSR training
program suggested by some observations Mednick had made about
the characteristic GSR activity recorded in schizophrenia and
in individuals with varying burdens of ancestral schizophrenia.
GSR modulation training was used with these subjects under the
general hypothesis that catastrophic autonomic nervous system
reactions are reflected in the GSR and are centrally associated
with the maintenance of active symptomatic schizophrenia. The
results we obtained after a three year follow-up interval justify
the view that the arousal of anxiety, which was modified in the
treatment program, may be more of a causal factor (rather than
an effect) in schizophrenic symptomatology. The results also hold
out some hope that some schizophrenic symptoms are susceptible
to suitably designed biofeedback training. Moreover, theresults
with our criminal population suggests that a less intense but
similar anxiety system is at the root of the criminial character
and thatcorrection of criminals demands reduction of their autonomic
arousaland especially reduction of their physiologically determined
anxiety.
CRIMINAL
OFFENDERS
In
our first pilot study with offenders, the subjects were 77 incarcerated
criminal offenders all males, mostly displaying the most dangerous
types of offenses, who gave evidence on the DDT of deep-brain
epileptic events. Presumably their deep subictal events were underlying
their excitement-seeking actions, their 'blind' rages, their excessive
sex drive and other derangements which were involved in their
criminal conduct.
STERMAN'S
SENSORI-MOTOR RHYTHM (SMR)
By
the time we were ready to work intensively with criminals Quirk
had heard Sterman give a paper at the 1970 Biofeedback meeting
in Boston. He had discussed Sterman's procedures with him and
both of us had obtained an Autogenic Systems 120a electroencephalometer
feedback instrument and had begun training a variety of subjects
to produce sensori-motor rhythm activity (SMR). EEG activity istypically
described as delta, theta, alpha, beta (for 1/2-3, 4-7, 8-13,
>13). The biofeedback professional adds to this nomenclation
the term SMR deriving from Sterman's discovery that while 13 Hz
is dominant through the brain, training 13 along the Fissure of
Roland results in resistance to epileptic seizure - even that
produced by toxins such as hydrazine (an normally infallible producer
of seizures of fatal intensity). The amplitude of 13 Hz is notoriously
small in epileptics over the RolandicFissure (aka Sensorimotor
Strip or sulcus centralis).
We
began to train SMR using the 120a Autogen version of the EEG.
All of our subjects were also trained to produce an increase in
skin resistance (GSR) and skin temperature using the SCARS method
we had developed a decade before and used with a wide variety
of scores of subjects, including our hospitalized female chronic
patients.
NO
PRODROME
It
is worth repeating that EEG anomalies are notfrequently observed
in patients with these deep-brain sub-ictal or complex-partial
ictal events. It is also worth repeating that no recognizable
warning events tend to occur. In fact, neurological lore is that
this kind of seizure requires an average of six nocturnal extended
EEG runs in an awake subject using nasal pharyngeal leads where
the electrodes are placed close to the base of the brain up through
the nose and the pharynx.
The
fact that the subject has no EEG anomalies means that the therapist
cannot depend on training which suppresses unwanted signals in
the EEG. It also means that the subject cannot establish a cue
to aid him to learn how to enhance his SMR or to normalize his
GSR.
DEPENDENT VARIABLES
During
the training phase of these studies on felons, the behaviour subjected
to operant training was sensorimotor rhythm (SMR) in the EEG,
the temperature recorded at the left index finger, and the galvanic
skin resistance (GSR). In all of the studies of felons, both of
these responses were recorded and shaped. This is unlike the work
with the chronic hospitalized psychotic patients who were only
trained by GSR and temperature.
SMR
was recorded on an Autogenic Systems 120a electroencephalo-meter.
Since absolute SMR values were less of interest than changes in
the strenghth of the signal at 13 Hz, we placed the electrodes
at C3 and C4. The limits we adopted and set in the equipment to
represent SMR activity were 12 to 14 Hz between 10 to 30 microvolts.
It
should be remarked that these values for amplitude (10-30 uV)
will appear to be high to those who use equipment manufactured
in the 1990s. An equivalent amplitude limit on contemporary equipment
would be closer to the 2 to 10 microvolt range. We restricted
the amplitude in this way in order to limit equipment response
to high amplitude artifacts in the low frequencies.
The
120a Autogen EEG does not have an Inhibit command, so that so-called
'inhibits' have to be all of that activity which is excluded by
the setting for reinforcement. The 120a is supplied with verniers,
that is knobs which are infinitely adjustable, and so lend themselves
to the behaviorist technique of 'shaping'. We shaped the frequency
down from a window opened to 10 Hz through 16 Hz and slowly moved
the verniers to a setting of 12 Hz through 14 Hz, viz. 13 Hz.
The amplitude remained fixed thoughout training. I have been slightly
flabbergasted to receive telephone calls asking me to explain
what I mean by "shaping". One shapes the behavior from
broad limits to narrow. The dolphin is rewarded for lifting his
head from the water, then for lifting higher, andall the way up,
then backward and finally for a flip. "Shaping" is the
fundamental principle of learning theory and technique.
We
now call the method, using temperature biofeedback training, GSR
feedback training (in Mary Cover Jones' model of successively
more stressful stimuli) and EEG training of the SMR, "Quirk's
modification of Sterman's (1970) neurotherapy". An aside
to those of you who bill insurance, when the insurance officer
says "experimental" look horrified and say "1970!??".
The
feedback used by Quirk was a frequency modulated whistle that
occurred discontinuously and contingently upon this SMR activity;
the machine will also utter a white "shhhhhh" sound
and this option was used by von Hilsheimer.
In
the typical case, the percent of the time in which this SMR activity
occurred tended to average around 10% during the first few half-hour
training sessions, and to rise to 45% to 55% during the last training
sessions. I should say that today I urge practitioners to shape
the behavior so that the client is achieving more than 85% feedback
success. This arises from the consensus of the practitioners I
have sampled. GSR was recorded on a locally constructed treatment
unit. A digital display changes in thousands of ohms of skin resistance.
The SCARS procedure described above was followed in exactly the
same manner as with the hospitalized schizophrenic females. However,
by the time we began working with felons, computer technology
had become more economical and user friendly and instead of slide
projections, in the more recent studies (1985 onward) computer
graphics were used rather than projected pictures from slides.
Any GSR increase ('less arousal') of 1K or more automatically
(a) entered the new GSR value in a memory chip for comparison
between chip and meter, and (b) changed the slide the subject
was watching or advanced the picture by computer. The slide contents
represented areas of emotional arousal selected for each subject
from her responses to a fears survey schedule. More individualization
was available once the pictures were computerized. Video feedback
(slide change) was provided discontinuously and contingently depending
upon successive increases in skin resistance. In the typical case,
GSR values tended to be in the range 50K to 100K. As reported
by Mednick, and consistent with our own experience in widely varying
treatment environments with widely varying populations diagnosed
as schizophrenic, the BSR of criminals was consistenly closer
to that of normals than was the BSR of schizophrenics. The latency
of change, the depth of change, and the lenghth or sluggishness
of time to change was markedly less in felons than in schizophrenics.
The
shape of graphed GSR change was sinusoidal especially in contrast
to the graphs of schizophrenics. The more schizoid the characteristics
of the GSR, the more severe the behavior and less positive the
response of the felons to treatment.
We
remarked that schizophrenics and felons tended to normalize their
GSR/BSR values as well as the intensity of response in the last
treatment sessions. The GSR values during the last treatment sessions
tended to vary from 150K to 550K ohms.
I
should also remark here that I am continually being asked that
ifschizophrenics have such a high BSR in contrast to normals,
"why do wetrain it up?" The real reason I do it is that
Quirk told me to do so; and that for more than 40 years I have
enjoyed success doing so. The BSRcontinually normalizes and comes
down when you continue to train it up.At least that is true in
all the clients I have observed in all that time. Presumably an
increase in ohms of resistance is an increase in the comfort of
the organism. Why such highly uncomfortable and dysphoric humans
as schizophrenic patients should have such enormously highskin
resistance is really unanswerable.
The
dependent measures for the main effects of the studies of felons
were re-occurrences of condition relevant behaviour during an
eighteen month follow-up interval after discharge. Offenders were
followed up through their cumulative justice system offense records
a year and a halfafter release from the sentences in which they
were treated.
Results
(1)
The justice system records of the offenders accepted into this
biofeedback treatment programme were reviewed an average of a
year and a half after release from their treatment sentences.
Of the 17 offenders who received 0 to 4 half-hour training sessions
(i.e., essentially no treatment), 65% had been re-convicted of
criminal offenses. Of the 10 who had received 34 or more half-hour
training sessions, only 2 or 20% had been re-convicted of criminal
offenses. Intermediate amounts of training were found to be associated
with intermediate recidivism rates. Among these subjects, neither
follow-up interval nor age were related to recidivism rates.
STUDIES SUBSEQUENT TO
THE PILOT STUDY
The death of the senior author
(Quirk) in December of 1997 after he completed a second draft
of this paper, and read my third draft, made impossible our plans
for a leisurely reporting of a long (25 year) series of studies
done with SCARS and Quirk's Sterman treatment of a large (nearly
3000) number of jailed felons.
From
1970 until his retirement in 1995 Quirk continued to work at the
Ontario Correctional Institute near Toronto, Ontario with the
daily collaboration of his colleague at the OCI, Reg Reynolds.
Those of us with more mobile staff may envy the fact that Reynolds
and Quirk had lunch together every working day for more than 20
years. Quirk and Reynolds did significant work on Cognitive Behavior
Therapy with felons; and Quirk carried out the EEG studies essentially
enjoying the benign neglect of his nominal superiors. Reynolds
is Quirk's executor and has been most helpful in organizing and
mining Quirk's papers.
From
1970 through mid 1995 Quirk used the DDT method of examining felons,
appointing them for EEG and SCARS training. The actual work of
training was done by volunteers, including correctional guards,
college students, housewives and others.
In
a series of reports to the Corrections officials Quirk indicated
that the three year recidivism rate from the biofeedback unit
(using these techniques) had never been more than 45%, and in
some groups had fallen to 15%. Overall, among nearly 3000 felons
trained by Quirk's volunteers, the rate of recidivism was consistently
close to 15%. In every comparative group, the rate of success
of the biofeedback group was significantly superior to the results
in the Ontario Correctional Institute (OCI) in groups which did
not use biofeedback. It should be noted thatthe OCI was generally
known as "the best jail in North America".
If
you consider the violent crimes which the felons trained by Quirk
would have committed and did not because of their training at
OCI the Quirk Sterman protocol has saved thousands of lives.
Discussion
It was our intent originally
only to discuss the results of the initial pilot study and then
go on to the 25 year record which followed this study. The original
study did suggest that even some prodromeless conditions would
be susceptible to biofeedback interventions using appropriate
applications. Adequate results will be obtained if the training
is structured to foster the establishment of a new and stable
habit to respond in healthy ranges of appropriately selected physiological
responses. Lasting and self strengthening resistance to symptoms
may be possible.
In
these studies, the physiological responses monitored were considered
to be operant responses and the responses were subjected to reinforcements
to shape new habits independent of voluntary control or understanding.
It is assumed that the habits thus developed might well be self-strengthening
since, if properly selected, they should eventuate in self-reinforcement
by enhancing the efficiency of the person's functioning.
The
other issue this report reflects is the physiological responses
we monitored and trained. In many conditions, there is a recordable
physiological response that varies with the symptoms. It is probably
relevant only to the preferences of the particular investigator
whether it is the physiology or it is the behaviour drives, that
controls or causes the other. Regardless of the 'causal' relationships
involved, treatment might be effected by trained modification
of the most convenient response to monitor. It is often easier
to monitor the physiological response, and by training changes
in the physiological reaction we may be able to change the symptoms
of the patient. Why else would we do it? Oddly enoughin psychology
sometimes whe change a response just to see if we cantrain it.
Fortunately, the results here indicated that we were obtaininga
more useful result. Modifying the skin resistance changed the
behavior - either the felons didn't commit more crimes, or at
least theyweren't caught and arrested doing them.
In
the second study we talked about in this paper deep-brain epilepsy
was detected in a subset of relatively dangerous offenders. In
spite of the fact that EEG anomalies were not readily detected
among these subjects for treatment modification, under the hypothesis
that Sterman's method for SMR training offers a general treatment
for epilepsy, we tried using Sterman with these offenders after
we had done GSR training. We obtained stable and large effects
which reduced the subsequent criminal behaviour of our felons.
Finally
since study began 25 years of successful treatment of 2776 violent
felons who had sub-ictal indications on a paper and pencil test,
the DDT. The conclusions we drew are vital and indicate that the
combination of temperature, GSR-SCARS training forming Von Quirk's
Sterman method for EEG training are robust and effective training
methods which have immediate relevance for today's population
of criminals and mental health patients.
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